[1] MAYER-HAMBLETT N,RETSCH-BOGART G,KLOSTER M,et al.Azithromycin for early Pseudomonas infection in cystic fibrosis.the OPTIMIZE randomized trial[J].Am J Respir Crit Care Med,2018,198(9):1177-1187. [2] MONOGUE M L,STAINTON S M,BAUMMER-CARR A,et al.Pharmacokinetics and tissue penetration of ceftolozane-tazobactam in diabetic patients with lower limb infections and healthy adult volunteers[J].Antimicrob Agents Chemother,2017,61(12):e01449-e01417. [3] GIRMENIA C,BERTAINA A,PICIOCCHI A,et al.Incidence,risk factors and outcome of pre-engraftment gram-negative bacteremia after allogeneic and autologous hematopoietic stem cell transplantation:an Italian prospective multicenter survey[J].Clin Infect Dis,2017,65(11):1884-1896. [4] 于犇犇,汪璐璐,贺文涛,等.2013—2017年ICU感染病原菌分布及耐药趋势分析[J].中国抗生素杂志,2019,44(10):1198-1202. [5] KAMOSHIDA G,YAMADA N,NAKAMURA T,et al.Preferential selection of low-frequency,lipopolysaccharide-modified,colistin-resistant mutants with a combination of antimicrobials in Acinetobacter baumannii[J].Microbiol Spectr,2022,10(5):e0192822. [6] ZHAO X,DRLICA K.Restricting the selection of antibiotic-resistant mutants:a general strategy derived from fluoroquinolone studies[J].Clin Infect Dis,2001,33(Suppl 3):S147-S156. [7] 黄世杰. 复方头孢他啶/阿维巴坦治疗医院获得性肺炎的Ⅲ期临床试验获阳性结果[J].国际药学研究杂志,2016,43(5):828. [8] 梅清,耿士窠,房晓伟,等.头孢他啶-阿维巴坦联合多黏菌素E对广泛耐药铜绿假单胞菌的体外抗菌活性[J].中华危重病急救医学,2019,31(10):1212-1218. [9] Performance Standards for Antimicrobial Susceptibility Testing,26th Edition:CLSI M100-S26[S].Clinical And Laboratory Standards Institute [clsi],. [10] FDA.Avycaz (ceftazidime-avibactam) package insert[EB/OL].(2016-03).http:∥www.Accessdata.Fda.gov/drugsatfda_docs/label/2015 /206494s000lbl.Pdf. [11] EHMANN D E,JAHIĆ H,ROSS P L,et al.Avibactam is a covalent,reversible,non-β-lactam β-lactamase inhibitor[J].Proc Natl Acad Sci USA,2012,109(29):11663-11668. [12] CUI J C,LIU Y N,WANG R,et al.The mutant selection window in rabbits infected with Staphylococcus aureus[J].J Infect Dis,2006,194(11):1601-1608. [13] WANG H H,CHEN Y,STRICH J R,et al.Rapid detection of colistin resistance protein MCR-1 by LC-MS/MS[J].Clin Proteom,2019,16(1):1-10. [14] GARRIGÓS C,MURILLO O,LORA-TAMAYO J,et al.Fosfomycin-daptomycin and other fosfomycin combinations as alternative therapies in experimental foreign-body infection by methicillin-resistant Staphylococcus aureus[J].Antimicrob Agents Chemother,2013,57(1):606-610. [15] CHEN H W,GUO X C,XIE D C,et al.A clinical study on the use of intraventricular polymyxin B supplemented by continuous external ventricular drainage in the treatment of drug-resistant gram-negative bacilli intracranial infection[J].Infect Drug Resist,2020,13:2963-2970. [16] TAMM M, EICH C, FREI R, et al.Inhaled colistin in cystic fibrosis[J].Schwz Med Wochenschr, 2000, 130(39):1366-1372. [17] LI J,TURNIDGE J,MILNE R,et al.In vitro pharmacodynamic properties of colistin and colistin methanesulfonate against Pseudomonas aeruginosa isolates from patients with cystic fibrosis[J].Antimicrob Agents Chemother,2001,45(3):781-785. [18] NATION R L,GARONZIK S M,THAMLIKITKUL V,et al.Dosing guidance for intravenous colistin in critically-ill patients[J].Clin Infect Dis,2017,64(5):565-571. [19] WEI W J,YANG H F.Synergy against extensively drug-resistant Acinetobacter baumannii in vitro by two old antibiotics:colistin and chloramphenicol[J].Int J Antimicrob Agents,2017,49(3):321-326. [20] MIKHAIL S,SINGH N B,KEBRIAEI R,et al.Evaluation of the synergy of ceftazidime-avibactam in combination with meropenem,amikacin,aztreonam,colistin,or fosfomycin against well-characterized multidrug-resistant Klebsiella pneumoniae and Pseudomonas aeruginosa[J].Antimicrob Agents Chemother,2019,63(8):e00779-e00719. [21] MOSKOWITZ S M,ERNST R K,MILLER S I.PmrAB,a two-component regulatory system of Pseudomonas aeruginosa that modulates resistance to cationic antimicrobial peptides and addition of aminoarabinose to lipid A[J].J Bacteriol,2004,186(2):575-579. [22] MCPHEE J B,BAINS M,WINSOR G,et al.Contribution of the PhoP-PhoQ and PmrA-PmrB two-component regulatory systems to Mg2+-induced gene regulation in Pseudomonas aeruginosa[J].J Bacteriol,2006,188(11):3995-4006. [23] SCHUREK K N,SAMPAIO J L M,KIFFER C R V,et al.Involvement of pmrAB and phoPQ in polymyxin B adaptation and inducible resistance in non-cystic fibrosis clinical isolates of Pseudomonas aeruginosa[J].Antimicrob Agents Chemother,2009,53(10):4345-4351. [24] FERNÁNDEZ L,GOODERHAM W J,BAINS M,et al.Adaptive resistance to the last hope antibiotics polymyxin B and colistin in Pseudomonas aeruginosa is mediated by the novel two-component regulatory system ParR-ParS[J].Antimicrob Agents Chemother,2010,54(8):3372-3382. [25] FERNÁNDEZ L,JENSSEN H,BAINS M,et al.The two-component system CprRS senses cationic peptides and triggers adaptive resistance in Pseudomonas aeruginosa independently of ParRS[J].Antimicrob Agents Chemother,2012,56(12):6212-6222. [26] LEE J Y,CHUNG E S,NA I Y,et al.Development of colistin resistance in pmrA-,phoP-,parR- and cprR-inactivated mutants of Pseudomonas aeruginosa[J].J Antimicrob Chemother,2014,69(11):2966-2971. |